ASPCR Detection of the Point
Mutation N291S in the LPL Gene
in Homo sapiens DNA with Familial Combined Hyperlipidemia
The Fantastic Four
Jane Dickson, Meghna Dhir, Luke Friedli, Tim Oja
Abstract
Familial Combined Hyperlipidemia (FCH) is caused by mutations in the
lipoprotein lipase (LPL) gene. The N291S mutation is a particularly common
mutation present in FCH patients (Hoffer et al, 1996).
This mutation changes the 291st amino acid from asparagine
to serine, resulting in protein malfunction while preventing the correct breakdown
of lipoproteins. The purpose of our research was to develop a method of
detecting this mutation at the molecular level. A PCR diagnostic assay was
designed to determine the presence of the N291S mutation in DNA samples
extracted from human adherent epithelial cells. Two forward primers (WFP and
MFP) and a reverse primer (RP) were developed using the Yaku
method to anneal to either wildtype or mutant DNA (Yaku et al, 2008). We hypothesized that the inclusion of an
intentional mismatch at the third base pair from the 3’ end of each forward
primer would allow for the detection of the N291S mutation using PCR and gel
electrophoresis. Following PCR, the amplified products were analyzed by gel
electrophoresis to determine the presence of N291S. Our PCR diagnostic assay
was unsuccessful in producing a 622bp band using the mutant forward primer with
mutant DNA and the wildtype forward primer with wildtype DNA, when paired with a standard designed reverse
primer amplifying the target region. As a control, two primers from a similar
study published in Arteriosclerosis,
Thrombosis, and Vascular Biology unsuccessfully detected the N291S
mutation. The members of this research team followed the recommended diet and
exercise regimens for a FCH patient to understand the effects of FCH on a
college student. We analyzed off-campus food budgeting and on-campus dining
accommodations and found that the diet required an average of $20.05 more per
week while MSU is making adequate accommodations for students with FCH. Regular
blood pressure testing and lipid panels evaluated the effect of these lifestyle
changes. We found that our blood pressure and lipid levels decreased to or
maintained healthy levels of 120/80 and 200mg/dL,
respectively. Our diagnostic assay holds significance because early detection
and treatment of FCH leads to the most effective results (Murphy 1974). Our
sociological experiment carries important implications because it sought to
recognize the challenges faced by FCH patients.
Figure1: Non-Specific Binding
using GoTaq Green Master Mix from Promega.
The GoTaq Green Master mix cocktail creates an
ideal stringency by containing predetermined volumes of Taq
polymerase, dNTP’s, a reaction Buffer, and MgCl2. In order to test our designed primers, we
added 1 µl of WFP, 1 µl RP, 2 µl S9 cells, and 21 µl of H2O to 25 µL
of the Master mix. The cocktail was run in the PCR
machine with a denaturation temperature of 94˚C
for an intial cycle of 5 minutes and for 30 seconds
for each following cycle. An annealing
temperature of 48˚C was used for 1 minute, the extension was run at 72˚C
for 1 minute for each cycle and the final elongation was run for 5 minutes.
This cocktail was to determine that our primers could act as a positive control
for wildtype DNA.
In lane 2, there was non-specific binding with a stronger band at the
desired band length of 622 base pairs.
The possible non-specific binding was most likely due to the low
annealing temperature. However, the
greater intensity between the 650 and 500 base pair bands of the 1 Kb Plus
Ladder indicates that more favorable results of the desired band at the length
of 622 base pairs would have occurred had the temperature been slightly
increased.
Figure 3: Non-Specific Binding
for positive controls of wildtype testing. The first letter for each lane
is the type of DNA used, M for mutated DNA and W for wildtype. The second letter refers to the primers used,
P for the control primers and W for the MFP and RP. The temperatures then indicate the annealing
temperature chosen. The cocktails were initially denatured at 94 ºC for 5
minutes and then at 94 ºC for 30 seconds for each of the 30 cycles. They were then annealed for 1 minute at 30 ºC,
45 ºC, or 50 ºC, and extended for 1 minute at 72 ºC with a final extension of 5 minutes at 72 ºC. The highlighted regions emphasize the possible
non-specific binding. The non-specific
binding in lane 3 of gel B encouraged the increased temperature for gel A. The
region in the smaller box of lane 3 of gel A leads to
believe that in the raised annealing temperatures the desired band would be
created in the future. Additionally at
the bottom of this lane there are possible primer dimers;
however, unsupported due primers not being run alone. The cocktail in lane 1 of gel A consisted of a negative control because it was the control
primers from the Zhang et al, paper for mutated DNA run with wildtype DNA. Future tests are needed to solidify these
findings.
Discussion
Background
Familial Combined Hyperlipidemia is a hereditary disorder characterized by
high levels of LDL, cholesterol, and, triglycerides in affected individuals (Brunzell et al, 1983).
These symptoms are the result of abnormalities in lipoprotein
metabolism, which is caused by a defective form of the enzyme lipoprotein
lipase (LPL) (Reymer et al, 1995). The N291S mutation is one of many that can
occur in the LPL gene, resulting in a single base pair substitution from
adenine to guanine (Zhang et al, 1995).
This mutation is common to a high frequency of FCH patients (Reymer et al, 1995).
In our study, we attempted to design a PCR diagnostic test that can
conclusively confirm whether or not an individual carries the N291S mutation.
As a second goal, our experiment investigated the impact that FCH has on a
college student. We focused on how a college student could accommodate the
necessary diet on and off campus. In order to do this, the researchers followed
a diet and exercised regularly much like an FCH
patient would be required (Cabezas et al, 1993).
Original
Predications
We hypothesized that our designed
primers would differentiate between wildtype and
mutant DNA during the PCR, which in turn would result in specific band patterns
following gel electrophoresis. In the
case of wild-type DNA, the WFP was predicted to produce a 622bp band when
paired with the RP, while the MFP was predicted to show no band after gel
electrophoresis. The band would be 622 bps long because the 5’ ends of the
forward primers are 622 bp’s
away from the 5’ end of the RP. This was
because the single intentional mismatch on the third base pair from the 3’ end,
incorporated based on the Yaku method, on the WFP
should not affect annealing and subsequent extension of the primer, provided
that our concentrations and annealing temperatures were adequate (Yaku et al, 2008).
In comparison, the MFP included both the intentional mismatch on the
same base pair and the mutation on the last base pair or the 3’ end, therefore
preventing extension of the primer because there would be three base pairs on
the 3’ end of the primer, which do not anneal due to the two mismatches. In the case of mutant DNA, the MFP was
predicted to produce a 622bp band, while the WFP was predicted to show no band.
Both MFP and WFP contain the intentional mismatch; however, WFP contains an
additional mismatch at the site of the mutation that will prevent extension of
the primer (Yaku et al, 2008). We hypothesized that
MSU provides the necessary resources and accommodations that would allow a
student with FCH to have a college experience similar to that of an unaffected
student. We predicted that the health
variables being measured, which included blood pressure, cholesterol level,
triglycerides, HDL, and LDL levels, would show a steady decrease to healthier
levels for two reasons (Wong et al, 2000).
The first comes from our knowledge that a wide variety of foods are
served on campus and that the cafeterias would provide healthier options to fit
within our diet (Shannon et al, 1997).
The second reason involves the availability exercise facilities on
campus.
Genomic
Purification
After completing genomic
purification, a purity ratio of 2.176 was found through a spectrophotometer,
showing more DNA then protein was extracted.
Results and Ultimate
Findings
After a total of six different
PCR diagnostic trials, PCR detection of the N291S mutation was unsuccessful. Additional
trials are required in order to detect the mutation in both the wildtype and mutant DNA (Roux, 2009).
Successful detection of the Rz gene of bacteriophage lambda
was utilized to determine the cocktail ingredients for the wildtype
DNA cocktail that included the wildtype DNA forward
and reverse primers. The other cocktail was adapted from a previously successful
PCR detection of the N291S mutation (Zhang et al, 1995). It was found that the
paper cocktail used more reaction buffer than the lambda cocktail. After
running both cocktails simultaneously for all the trials, it was apparent that
non-specific binding occurred in the lanes that contained the lambda cocktail.
This can be attributed to the fact that excess buffer can affect the optimal
annealing temperatures (Roux, 2009).
During trial 1, we ran both
cocktails at the annealing temperatures 48°C and 51°C, which showed that our targeted region was not
amplified. Thus, we decided to drop the temperatures to 35°C and 40°C and slowly increase the
temperatures as necessary. Temperatures were then increased to 30°C and 45°C. In the lanes that were ran at
45°C, there appeared to be
non-specific binding. Non-specific binding occurs when the primers anneal to
multiple points on the DNA (Ausubel, 1987).
A method to reduce non-specific binding is to raise the annealing temperature (Roux, 2009).
Our lasts gels were then run at 46°C and 48°C and continued to show slight non-specific binding.
A very faint band appeared around the predicted band length of 622 base pairs
for the paper cocktail using wildtype DNA and wildtype primers, however another
trial is needed to confirm the presence of the band. However, the lane also
showed possible primer dimer, which could be
attributed to incorrect primer design, causing the primers to anneal to each
other (Cha et al, 1993).
After several failed attempts to
amplify the targeted region, it was found that the wildtype
and mutant forward designed primers did not have a 40-60% G/C content, or G/C
clamp. G/C bonds are very important in primers due to three hydrogen bonds
between the nitrogenous bases (Roux, 2009). The three hydrogen bonds provide
strength and promote efficiency during the annealing phase in PCR (Ausubel, 1987).
From some recalculation, it was also apparent that the melting temperatures for
each the forward and reverse primers for both mutant and wildtype
were calculated incorrectly (Roux,
2009). The
temperatures were all a few degrees lower than they should have been. This can
be a possible reason for the non-specific binding that is visible gel trials 4,
5, and 6 that unsuccessfully amplified the targeted region. To strengthen our experiment, we performed a
negative control (Cha et al, 1993). We tested the wildtype
DNA with the CRP and CFP. It was predicted that no bands would appear on the
gel. However, after running the gel, it was found that some faint bands did
appear in multiple spots on the gel. This could be attributed to a flaw in
primer design, causing primers to anneal to random places on the DNA (Roux, 2009).
In terms of our sociological experiment, our hypothesis was supported and we
found that a college student at MSU would be able to make sufficient
accommodations to follow a healthy lifestyle. No significant decrease was shown
for blood pressure. Our lipid profile test, showed a decrease in overall
cholesterol for each member, but varied for the LDL, HDL, and triglyceride
levels. Variation can be attributed to the fact that this diet only lasted four
weeks, and more stable results are usually seen over a longer period (Cabezas et al, 1993). It was also found an average of 45%
of the foods were consumable on campus.
For someone living off campus, it was found that more money was spent
and fewer items were brought at each grocery trip, due to the fact that
healthier options are more expensive (Drewnowski et al,
2005).
Future Directions
With an unsuccessful PCR assay,
many parts of the experiment could be improved. The first three trials of gels
showed no bands at all. After much research, it was found that the annealing
temperatures were calculated incorrectly. To fix this problem, we dropped the
annealing temperatures to 30°C
and then raised it to try to find the optimal annealing temperature. After
raising the temperatures, we found non-specific binding appeared in many of the
lanes (Figure 3). In order to reduce non-specific binding, the temperature
should be raised by 2°C each
time and the duration of the annealing and extension steps should be shortened
(Cha el at, 1993). Also, the initial denaturation time could be lengthened to increase the
likelihood that the template DNA is fully denatured (Pal et al, 1991). The denaturation time that we used was five minutes; this could
be increased to seven. Non-specific
binding can also be reduced by varying the amounts of Mg2+ added to
each cocktail (Roux, 2009). Lower Mg2+
concentration can reduce non-specific binding because Taq
polymerase is an enzyme that depends on magnesium in order to provide
thermo-stability (Cha el at,
1993). Possible
primer dimer was also visible (Figure 3). To refute
or support this, both the primers should be run together (Roux,
2009). If given more
time, we would redesign the primers so that each would have an increased G/C
content to strengthen bonding. We would use Primer-BLAST,
and Oligo Design for assistance. After some research,
it was found that “Hot Start” PCR could improve our results by reducing the
chances of primer dimer and non-specific binding (Cha
el at, 1993). Hot Start PCR is a technique that raises the temperature above
the annealing temperature of the reaction components before the taq is added, thus increasing specificity (Roux, 2009).
Another method to reduce non-specific binding is called nested PCR, which uses
two sets of primers in two successive PCR trials (Roux, 2009).
The product of the first pair of primers is then used in the second PCR, with
has a set of primers that are different from the first reactions. Therefore,
this increases the specificity of the amplified DNA, and reducing non-specific
binding (Pal et al, 1991). Mutant DNA would be obtained from Dr. Paul Reymeror from the University of Illinois would
allow for further testing to amplify the N291S mutation with respective
primers.
In order to improve our
sociological assay, a long-term diet could be adapted for three months and a
deeper analysis of all the different meal times could be taken, which includes
looking at both breakfast and lunch.
Still-frame video: http://www.youtube.com/watch?v=fzS9oeqNZvw.